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Study identifier: NCT07695090 Synced from ClinicalTrials.gov · July 29, 2026
● Recruiting

Lumacaftor Yields Reversal of Impaired Cerebral Blood Flow in Heart Failure Patients

Condition: Heart Failure With Reduced Ejection Fraction  ·  Sponsor: Qanatpharma AG

PhasePhase 2
Planned participants60
Who can joinAll sexes, 18 Years to no upper limit
Healthy volunteersNo

About this study

Cognitive impairment (CI) is highly prevalent in patients with heart failure with reduced ejection fraction (HFrEF), which has significant implications for disease management, quality of life and clinical outcomes. Currently, there are no specific treatments for CI aside from the current standard of care therapy for HF, making this a high unmet medical need. Impaired cerebral autoregulation is a proposed mechanistic factor that leads to cerebral hypoperfusion, ischemic damage and the development for CI. Preclinical data indicates that restoring CFTR-protein expression normalizes cerebral microvascular function and cerebral blood flow (CBF) in models of HF. The purpose of this study is to investigate whether CFTR-targeting therapy enhances cerebral perfusion and cognitive function in heart failure patients using the CFTR-corrector Lumacaftor.

This description comes directly from the study's public registry record.

Talk to the study team

Qanatpharma Clinical Program Manager  ·  1-877-308-4220  ·  info@qanatpharma.com

Always discuss trial participation with your own doctor first.

Locations (2)

St. Michael's HospitalToronto, Ontario, CanadaRecruiting
University Health Network (UHN) Peter Munk Cardiac CentreToronto, Ontario, CanadaNot Yet Recruiting

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Source record: clinicaltrials.gov/study/NCT07695090