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Study identifier: NCT00992901 Synced from ClinicalTrials.gov · July 28, 2026
● Recruiting

Role of Neural and Hormonal Regulation Factors on Insulin Secretion After Gastric Bypass Surgery

Condition: Post Bariatricsurgery · Hypoglycemia  ·  Sponsor: The University of Texas Health Science Center at San Antonio

PhaseEARLY_Phase 1
Planned participants160
Who can joinAll sexes, 18 Years to 65 Years
Healthy volunteersYes

About this study

RYGB (roux-en-y gastric bypass) has been reported to reverse type 2 diabetes (T2DM) immediately after surgery before any significant weight loss. In addition, a growing number of patients have been recognized with life-threatening hyperinsulinemic hypoglycemia several years following their surgery. While the mechanisms by which RYGB improves glucose metabolism or alters islet cell function in patients after RYGB are not understood, recent studies suggest that increased secretion of GI hormones, primarily glucagon-like peptide 1 (GLP-1), as well as alteration in neural activity may contribute to enhanced insulin secretion in general, and to a greater extent in patients with hypoglycemia. The proposed research is designed to address the role of RYGB on insulin secretion by evaluating the contribution of stimulatory factors (neural and GI hormone) on islet cell function and the islet cell responsiveness to the physiologic stimulatory factors, in RYGB patients with and without hypoglycemia and non-operated controls.

This description comes directly from the study's public registry record.

Talk to the study team

Marzieh Salehi, MD MS  ·  210-567-6691  ·  salehi@uthscsa.edu

Jennifer Foster, MSN  ·  210-450-8696  ·  fosterj6@uthscsa.edu

Always discuss trial participation with your own doctor first.

Locations (2)

Texas Diabetes Institute - University Health SystemSan Antonio, Texas, United StatesRecruiting
South Texas Veterans Health Care SystemSan Antonio, Texas, United StatesRecruiting

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Source record: clinicaltrials.gov/study/NCT00992901